International Journal of Cardiology
Volume 111, Issue 3 , Pages 442-449, 28 August 2006

Elevated troponin level is not synonymous with myocardial infarction☆☆☆

  • Nitin Mahajan

      Affiliations

    • Department of Medicine, Maimonides Medical Center, Brooklyn, NY 11219, USA
    • Corresponding Author InformationCorresponding author. Tel.: +1 347 277 6497.
    • ,
  • Yatin Mehta

      Affiliations

    • Department of Medicine, Maimonides Medical Center, Brooklyn, NY 11219, USA
    • ,
  • Malcolm Rose

      Affiliations

    • Department of Medicine, Maimonides Medical Center, Brooklyn, NY 11219, USA
    • ,
  • Jacob Shani

      Affiliations

    • Department of Cardiology, Maimonides Medical Center, Brooklyn, NY 11219, USA
    • ,
  • Edgar Lichstein

      Affiliations

    • Department of Medicine, Maimonides Medical Center, Brooklyn, NY 11219, USA

Received 9 May 2005; received in revised form 6 August 2005; accepted 29 August 2005. published online 15 November 2005.

To access this article, please choose from the options below

Login to an existing account or Register a new account.

  • Purchase this article for 31.50 USD (You must login/register to purchase this article)

    Online access for 24 hours. The PDF version can be downloaded as your permanent record.

  • Subscribe to this title

    Get unlimited online access to this article and all other articles in this title 24/7 for one year.

  • Claim access now

    For current subscribers with Society Membership or Account Number.

  • Visit SciVerse ScienceDirect to see if you have access via your institution.

☆ Supported in part by Maimonides Research Grant.

☆☆ This is the largest series on retrospective analysis of patients with elevated troponin levels. We believe that this study elucidates the etiology and pathophysiology of elevated cardiac troponin I in patients with normal coronary arteries on angiography. Elevated troponins reflect myonecrosis as a result of imbalance between myocardial oxygen supply and consumption. Acute myocardial infarction is a clinical diagnosis. Elevated troponin levels should be interpreted appropriately based on underlying clinical setting. This study is a retrospective study, and hence did not involve any patient contact. Also, it had the approval of the Internal Review Board.

PII: S0167-5273(05)01162-9

doi:10.1016/j.ijcard.2005.08.029

International Journal of Cardiology
Volume 111, Issue 3 , Pages 442-449, 28 August 2006

Article Tools