International Journal of Cardiology
Volume 135, Issue 2 , Pages 240-242 , 26 June 2009

Caucasian patients suffering from coronary vasospastic angina have an intact peripheral endothelium-dependent vasoreactivity

  • Ali Yilmaz

      Affiliations

    • Division of Cardiology, Robert-Bosch-Hospital, Stuttgart, Germany
  • ,
  • Matthias Vöhringer

      Affiliations

    • Division of Cardiology, Robert-Bosch-Hospital, Stuttgart, Germany
  • ,
  • Anastasios Athanasiadis

      Affiliations

    • Division of Cardiology, Robert-Bosch-Hospital, Stuttgart, Germany
  • ,
  • Peter Ong

      Affiliations

    • Division of Cardiology, Robert-Bosch-Hospital, Stuttgart, Germany
  • ,
  • Rimma Merher

      Affiliations

    • Division of Cardiology, Robert-Bosch-Hospital, Stuttgart, Germany
  • ,
  • Dieter Ratge

      Affiliations

    • Division of Clinical Chemistry, Robert-Bosch-Hospital, Stuttgart, Germany
  • ,
  • Cornelius Knabbe

      Affiliations

    • Division of Clinical Chemistry, Robert-Bosch-Hospital, Stuttgart, Germany
  • ,
  • Udo Sechtem

      Affiliations

    • Division of Cardiology, Robert-Bosch-Hospital, Stuttgart, Germany
    • Corresponding Author InformationCorresponding author. Robert-Bosch-Krankenhaus, Auerbachstraße 110, 70376 Stuttgart, Germany. Tel.: +49 711 8101 3456; fax: +49 711 8101 3795.

Received 28 November 2007 ,Accepted 20 January 2008.

References 

  1. Anderson TJ, Uehata A, Gerhard MD, et al. Close relation of endothelial function in the human coronary and peripheral circulations. J Am Coll Cardiol. 1995;26:1235–1241
  2. Motoyama T, Kawano H, Kugiyama K, et al. Flow-mediated, endothelium-dependent dilatation of the brachial arteries is impaired in patients with coronary spastic angina. Am Heart J. 1997;133:263–267
  3. Motoyama T, Kawano H, Kugiyama K, et al. Flow-mediated, endothelium-dependent dilatation of the brachial arteries is impaired in patients with coronary spastic angina. Am Heart J. 1997;133:263–267
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  9. Motoyama T, Kawano H, Kugiyama K, et al. Flow-mediated, endothelium-dependent dilatation of the brachial arteries is impaired in patients with coronary spastic angina. Am Heart J. 1997;133:263–267
  10. Anderson TJ, Uehata A, Gerhard MD, et al. Close relation of endothelial function in the human coronary and peripheral circulations. J Am Coll Cardiol. 1995;26:1235–1241
  11. Yasue H, Horio Y, Nakamura N, et al. Induction of coronary artery spasm by acetylcholine in patients with variant angina: possible role of the parasympathetic nervous system in the pathogenesis of coronary artery spasm. Circulation. 1986;74:955–963
  12. Kaski JC, Tousoulis D, Gavrielides S, et al. Comparison of epicardial coronary artery tone and reactivity in Prinzmetal's variant angina and chronic stable angina pectoris. J Am Coll Cardiol. 1991;17:1058–1062
  13. Okumura K, Yasue H, Matsuyama K, et al. Diffuse disorder of coronary artery vasomotility in patients with coronary spastic angina. Hyperreactivity to the constrictor effects of acetylcholine and the dilator effects of nitroglycerin. J Am Coll Cardiol. 1996;27:45–52
  14. Masumoto A, Mohri M, Shimokawa H, Urakami L, Usui M, Takeshita A. Suppression of coronary artery spasm by the Rho-kinase inhibitor fasudil in patients with vasospastic angina. Circulation. 2002;105:1545–1547
  15. Miyao Y, Kugiyama K, Kawano H, et al. Diffuse intimal thickening of coronary arteries in patients with coronary spastic angina. J Am Coll Cardiol. 2000;36:432–437

PII: S0167-5273(08)00429-4

doi: 10.1016/j.ijcard.2008.01.053

International Journal of Cardiology
Volume 135, Issue 2 , Pages 240-242 , 26 June 2009