Tako-Tsubo cardiomyopathy (TCM) or “apical ballooning syndrome” is characterized by
transient left ventricular apical wall motion abnormalities with chest symptoms, electrocardiographic
changes and minimal cardiac enzyme release in the absence of coronary artery disease
[
[1]
]. Despite its increasing interest, the underlying physiopathological mechanisms remain
unclear. Abnormal catecholamine dynamics related to physical or emotional distress
seems to play a major role in the pathogenesis of this cardiomyopathy inducing acute
myocardial stunning, with a predilection for the cardiac apex [
[2]
]. The most commonly presented clinical symptoms were chest pain and dyspnea but various
manifestations of TCM can be observed [
3
,
4
]. Arrhythmias have been described as an unusual complication of TCM, including ventricular
arrhythmia, atrial fibrillation, torsade de pointe and conduction tissue dysfunction [
[5]
]. The management of high-degree AV block during early presentation of TCM remains
difficult.Keywords
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References
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Article info
Publication history
Published online: July 12, 2014
Accepted:
July 5,
2014
Received:
May 9,
2014
Identification
Copyright
© 2014 Elsevier Ireland Ltd. Published by Elsevier Inc. All rights reserved.
